|
Bliss M, The
importance of accurate terminology in wound
care.
J Tissue Viability
2003:13; 180.
Nomeh B, Yager D,
Cohen K. Physiology of the chronic wound.
Clin Plast Surg
1998:25; 341-357.
Tomic-Canie M,
Agren M, Alvarez O. Epidermal repair and the
chronic wound; in the Epidermis in Wound
Healing. Ed., D. Rover, H. Mailbach.
CRC press, New
York, 2004, p 25.
Wysocki AB and
Grinnell F. Fibronectin profiles in normal and
chronic wound fluid.
Lab Invest
63:825-831, 1990.
Weckroth M, Vaheri
A, Lauharanta J, Sorsa T and Konttinen YT.
Matrix metalloproteinases, gelatinase and
collegenase in chronic leg ulcers.
J Invest Dermatol
106:1119-1124, 1996.
Herrick S,
Ashcroft G, Ireland G, Horan M, McCollum C and
Ferguson M. Up-regulation of elastase in acute
wounds of healthy aged humans and chronic venous
leg ulcers is associated with matrix
degradation. Lab Invest 77: 281-288, 1997.
Trengove NJ,
Stacey MC, MacAuley S, Bennett N, Gibson J,
Burslem F, Murphy G, and Schultz G. Analysis of
the acute and chronic wound environments: the
role of proteases and their inhibitors. Wound
Repair Regen 7:442-452, 1999.
Agren MS,
Eaglstein WH, Fergtuson MWJ, Harding KG, Moore
K, Saarialho-Kere U and Schultz GS. Causes and
effects of the chronic inflammation in venous
leg ulcers.
Acta Derm Venereol
(Stockh). Suppl 210, 3-17, 2000.
Saarialho-Kere UK,
Patterns of matrix metalloproteinase and TIMP
expression in chronic ulcers. Arch Dermatol Res
(Suppl.), 290, S47-S54, 1998.
Haslett C and
Henson P. Resolution of inflammation in the
Molecular and Cellular Biology of Wound Repair,
Clark RAF, ed. Plenum Press, New York, 1998, pp
143-168.
Folanga V. Growth
factors and chronic wounds, the need to
understand the microenvironment. J Dermatol
19:617-672, 1992.
Grinnell F, Ho CH,
Wysocki A. Degradation of fibronectin and
vitronectin in chronic wound fluid. Analysis
by cell blotting, immunoblotting and cell
adhesion assays. J Invest Dermatol 98:410-4126,
1992.
Grinnel F, Zhu M.
Identification of neutrophil elastase as the
proteinase in burn wound fluid responsible for
degradation of fibronectin. J Invest Dermatol
103:155-161, 1994.
Grinnel F, Zhu M.
Fibronectin degradation in chronic wounds
depends on the relative levels of elastase,
alpha 1-proteinase inhibitor, and alpha
2-macroglobulin.
J Invest Dermatol
106:355-341, 1996.
Harris IR, Yee KC,
Walters CE, et al. Cytokine and protease levels
in healing and non-healing chronic venous leg
ulcers. Exp Dermatol 4:342-349, 1995.
Wysocki AB,
Staiano-Coico L and Grinnel F. Wound fluid from
chronic leg ulcers contains elevated levels of
metalloproteinases MMP-2 and MMP-9, 1995.
J Invest Dermatol
101:64-68, 1993.
Rao CN, Ladin DA,
Liu YY, Chilukuri K, et al. ą1–Antitrypsin is
degraded and nonfunctional in chronic wounds but
intact and functional in acute wounds: the
inhibitor protects fibronectin from degradation
by chronic wound fluid enzymes.
J Invest Dermatol
105: 572-578, 1995.
Wysocki AB and
Grinnell F. Fibronectin profiles in normal and
chronic wound fluid.
Lab Invest
63:825-831, 1990.
Grinnell F, Ho CH,
and Wysocki AB. Degradation of fibronectin and
vitronectin in chronic wound fluid; analysis by
cell blotting, immunoblotting and cell adhesion
assays. J Invest Dermatol 98: 410-416, 1992.
Wysocki AB,
Staiano-Coico L, and Grinnell F. Wound fluid
from chronic leg ulcers contains elevated levels
of metalloproteinases MMP-2 and MMP-9.
J Invest Dermatol
101:64-68, 1993.
Lemat A, French L,
Blal L, et al. Organotypic cultures of
antologous hair follicle keratinocytes for the
treatment of recurrent leg ulcers.
J Am Acad Dermatol
2003:48; 207-14.
Curran M, Polskoi
G. Bilayered bioengineered skin substitute (Apligraf):
a review of its use in the treatment of venous
leg ulcers and diabetic foot ulcers. Bio Drugs
2002:16; 439-55.
Hanft J,
Surprenant M. Healing of chronic foot ulcers in
diabetic patients treated with a human
fibroblasts derived dermis.
J Foot Ankle Surg
2002:42; 291-99.
Jones I, Currie L,
Martin R. A guide to biologic skin substitutes.
Br J Plast Surg
2002:55; 185-93.
Badiavas E,
Paquette D, Carson P, Falanga V. Human chronic
wounds treated with bioengineered skin;
histologic evidence of host-graft interactions.
J Am Acad Dermatol 2002:46; 524-30.
Orgill D, Straus
F, Lee R. The use of collagen-GAG membranes in
reconstructive surgery. Ann N.Y. Acad Sci
1999:888; 233-48.
Hollander D, Stein
M, et al. Autologous keratinocytes cultured on
benzlester hyaluronic acid membranes in the
treatment of chronic full thickness ulcers. J
Wound Care 1999:8; 351-5.
Krejii N, Houng A,
Hansbrough J. Fibroblast sheets enable
epithelialization of wounds that do not support
keratinocyte migration.
Tissue Eng 1999:5;
555-62.
Khachemoune A,
Bello YM and Phillips TJ. Factors that
influence healing in chronic venous ulcers
treated with cryopreserved human epidermal
cultures. Dermatol Surg 28: 274-280, 2002.
Demling R, Nuzzoda
J et al. Small intestinal submucosa wound matrix
and full thickness venous ulcers. Wounds
16:18-22, 2004.
|
